Welcome! This blog contains research & information on lifestyle, nutrition and health for those with MS, as well as continuing information on the understanding of the endothelium and heart-brain connection. This blog is informative only--all medical decisions should be discussed with your own physicians.

The posts are searchable---simply type in your topic of interest in the search box at the top left.

Almost all of MS research is initiated and funded by pharmaceutical companies. This maintains the EAE mouse model and the auto-immune paradigm of MS, and continues the 20 billion dollar a year MS treatment industry. But as we learn more about slowed blood flow, gray matter atrophy, and environmental links to MS progression and disability--all things the current drugs do not address--we're discovering more about how to help those with MS.

To learn how this journey began, read my first post from August, 2009. Be well! Joan

Showing posts with label SIAPAV. Show all posts
Showing posts with label SIAPAV. Show all posts

Friday, January 4, 2013


CCSVI doppler diagnostics presented at vascular conference

January 4, 2013 at 2:09pm

The 34th Congress of the SIAPAV (Italian Society for Angiology and Vascular Medicine) http://www.siapav.it had an extensive review session of CCSVI diagnostics.  

There were three lectures presented by specialists in doppler ultrasound for CCSVI.  All of these specialists agree that CCSVI exists, it creates a delay of cerebral blood flow, hypoperfusion, and cerebral lesions, and there are very specific tests to measure this process.  

The specialists also stated that operators who are finding between 0-50% of CCSVI in people with MS are NOT utilizing correct doppler technique.  They stated that the SAME problem happened 35 years ago, when technicians began analysing the carotid arteries.  There is a very specific protocol that must be followed, or the results are inaccurate.  A lack of knowledge of the venous return system from the brain is part of this problem.

The fact that neurologists continue to ignore this clarion call from vascular specialists, and proceed with badly designed studies, is disturbing. There have been millions of dollars of wasted money and years of wasted time, pursuing studies that are of no value.

Below is the very specific review from this conference.
It is translated by google translate from Italian, and because if this, is not perfect.  But BNAC offers training for American doppler technicians, based on this protocol.  BNAC is the only center with a published US study that has worked with the experts in this doppler protocol.  None of the NMSS funded researcher utilized the complete and correct protocol.

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Refresher Course of the 34th Congress SIAPAV dedicated to CCSVI has provided an extensive updating on the diagnosis of chronic cerebrospinal venous insufficiency. The report that follows is intended as a reference for those interested in the clinical picture characterized by hemodynamic abnormalities and / or abnormalities of the refluxing veins from the Central Nervous System.

Three lectures were devoted to the physiology and pathophysiology of cerebral venous return (M. Mancini, Naples), which is essential for understanding the clinical phenomena, paintings echo-color-Doppler normal and pathological (G. Cacciaguerra, Catania), prerequisite for obtaining a correct diagnosis, limiting as much as possible the false negative and false positive and, finally, to the diagnostic protocol with the main criteria and / or accessories must be based on which the diagnosis (G. Arpaia, Vimercate).

Mancini has outlined the salient features of venous hemodynamics in the brain, illustrating the intracranial circulation time measured with echo contrast (landmarks carotid artery, jugular vein and thyroid) that are significantly delayed in CCSVI. From the point of view  the speaker presented the map of cerebral perfusion and the prevalence of MS plaques in the hypoperfused areas in the form of clinical progressive MS,  while the prevalence in areas hypoperfused would be greater in relapsing-remitting clinical form. This could explain the greater effectiveness of the correction hemodynamics in relapsing-remitting MS, compared to progressive MS.